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300519 RESULTS
David S. Bouvier
Laboratoire National de Santé, Luxembourg
Lisa Roys
Scientex ConferencesUnited States
Pan Sun
ZhenzhenwanChina
Parminder Singh
Postdoc United States
Iris Hardewig
Life Molecular ImagingGermany
Maria Wörheide
Stefka Gyoneva
United States
Miguel Angel Martinez Adrados
Spain
Qinglin Tang
Peking UniversityChina
APOE W5Ter
MUTATIONS APOE 45409896 GRCh37/hg19 rs777551553 G A Exon 2 Coding Predicted to abrogate production of ApoE as it introduces a stop codon in the signal peptide. PHRED-scaled CADD = 35. W5Ter Blood Lipids/Lipoproteins This variant was reported as a deletion of a thre
APOE T11S
MUTATIONS APOE 45409912 GRCh37/hg19 rs144354013 A T Exon 2 Coding Unknown, but predicted benign in silico (PHRED-scaled CADD = 0.6). T11S Alzheimer's Disease, Blood Lipids/Lipoproteins, Dementia This variant was examined in a study of dementia, including Alzhe
APOE A18T
MUTATIONS APOE 45411025 GRCh37/hg19 rs533904656 G A Exon 3 Coding Predicted to disrupt signal peptide cleavage and affect ApoE secretion. PHRED-scaled CADD = 22. A18T Alzheimer's Disease, Cardiovascular Disease This variant was reported in a small study in whi
APOE E21K
MUTATIONS APOE 45411034 GRCh37/hg19 rs121918392 G A Exon 3 Coding Increased receptor binding in cell-based LDL competition assay; no effect on heparin binding. E21K Multiple Conditions This variant has been found in multiple East Asian individuals with an abnormal
APOE A23V
MUTATIONS APOE 45411041 GRCh37/hg19 rs776242156 C T Exon 3 Coding Decreased plasma ApoE levels. A23V Alzheimer's Disease, Blood Lipids/Lipoproteins, Hyperlipoproteinemia Type IIa This variant was examined in a study of dementia, including Alzheimer’s disease (
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