Early Detection of Alzheimer Disease—A Virtual Town Hall Meeting
disease. Q: Does anyone believe PET imaging, perhaps with F-18 amyloid tracers, will be clinically
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Not Dead Yet: Estrogen Deserves Another Chance
that leuprolide increased longevity in rats by 18 percent with 6 months of treatment. Mark Smith Craig,
Alzheimer's: A Triple Whammy. Why are So Many Neurodegenerative Diseases Single, Double, or Triple Amyloidoses?
tauists and baptists finally shake hands? Trends Neurosci. 2002 Jan ;25(1):22-6. Abstract 18. Soto C.
Leaky Blood-Brain Barrier a Harbinger of Alzheimer's?
DCE-MRI scans of six healthy young people, 18 older folks who were cognitively normal, and 21 older adults
Suzanne Hendrix on Constructing Composites for Trials in Early Alzheimer’s
Have you ever been jolted wide awake by—biostatistics? That’s what happened on the last morning of the Clinical Trials on Alzheimer’s Disease conference held November 5-7 in Barcelona, Spain. At 8 a.m., as sleepy CTADeers slowly shuffled in, Suzanne Hendr
Isolation of Single Immunohistochemically Identified Whole Neuronal Cell Bodies from Post-Mortem Human Brain for Simultaneous Anlaysis of Multiple Gene Expression
for G3PDH and now we use primers for 18s ribosomal RNA and the rate of positives is roughly 50 percent
Alzheimer Presenilins in the Nuclear Membrane, Interphase Kinetochores, and Centrosomes Suggest a Role in Chromosome Segregation
Marc Paradis led this live discussion on 20 March 1998. Readers are invited to submit additional comments by using our Comments form at the bottom of the page. Paper under Discussion: Li JH, Xu M, Zhou H. Ma J. and Potter H. Alzheimer presenilins in the n
Pin1, Tau and the Cell Cycle in Alzheimer Disease
15;18:2801-2807. Abstract. 2. Campbell HD, Webb GC, Fountain S, Young IG. The human PIN1 peptidyl-prolyl cis/trans
Beta-Catenin and PS1
Chris Weihl, with Bruce Yankner, Benjamin Wolozin, Eddie Koo, and Kenneth Kosik led this live discussion on 13 August 1999. Readers are invited to submit additional comments by using our Comments form at the bottom of the page. Transcript: Live discussion
Generation of an Apoptotic Intracellular Peptide by γ-secretase Cleavage of Alzheimer's β-amyloid Precursor Protein
cleavage of poly [ADR-ribose] polymerase (PARP) by cell death protease known as caspases (18) also
The Role of ApoE in Alzheimer's Disease: An Alternative View
cultures. J Neurosci 18: 195-204. Abstract LaDu MJ, Falduto MT, Manelli AM, Reardon CA, Getz GS, Frail DE
Neural Stem Cells to the Rescue—Can Neuroreplacement Ever Become a Treatment for Alzheimer's?
and recovery of cognitive function, Behavioral and Brain Sciences, 18 (1995) 10-35. 0 Tuesday, July 9,
RNAi in Neurodegenerative Diseases—What's the Therapeutic Potential?
Medical School, Worcester, MA 01605 Zuoshang Xu led this live discussion on 18 March 2003. Readers are
Novel Substrates for BACE1 Beg the Question: What's the Biological Function of β-Secretase?
limits amyloid beta-peptide production. J Biol Chem. 2002 Feb 15;277(7):5637-43. Abstract 18.
Clioquinol Trial Postmortem: Does Blocking Metal-Aβ Interactions Work?
Chem. 2002 Oct 25;277(43):40302-8. Epub 2002 Aug 20. Abstract 18. Dong J, Atwood CS, Anderson VE,
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